Whether you live in an apartment or a house or simply would like to ensure your home is free of electromagnetic fields There are a variety of methods to reduce exposure. One of the most effective is to limit the use of electronic devices. You can also turn to EMF blocker paint to block EMF radiation from reaching your home. Another way to shield your house against EMF radiation would be to put up an RF shielding canopy. This is a fabric of net that contains EMF shielding and is used to block EMFs from entering rooms. Another option is to get your home fitted with an enclosure that is conductive. They are referred to as Faraday cages.
Numerous studies have demonstrated how the non-ionizing energy of RF produces antiproliferative properties in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed result from the deregulation the cancer stem cell. This may account for the long-term effects observed in patients suffering from advanced HCC. However, the mechanism of AM EMF's effects on patients with cancer is not evident.
Effects of AM RF EMFs on HCC tumor growth in vivo was studied in mice. The tumours were divided in three different groups. First, emf blockers that was unaffected to RF EMF. Second group members were exposed RF EMF at the same frequency to the one used by humans. Third group members were exposed to RF EMF with HCC-specific modulation frequencies. The effects of HCCMF on tumors was evaluated against that of RCF. The results indicated that cancers treated with HCCMF were significantly shrinking. However, the tumours treated with RCF didn't show evidence of shrinkage in the tumour.
The mechanism behind tumor-specific AM RF EMF might be due to the fact that cancer cells require Cav3*2 T-type voltage calcium channels for proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation on HCC cells is mediated by CACNA1H the protein which regulates the Ca2+ influx specific to tumors. emf blocker indicate that CACNA1H could have wider implications for the treatment and diagnosis of many cancers.
emf blocker in the control group were not exposed RF EMF, and were fed a normal mouse diet. The tumors of the HCCMF group were injected with Huh7 cells after they were five-seven weeks old. The tumors were removed when they showed excessive burden.
The tumours in the three groups showed different growth curves. The tumors treated with HCCMF showed a significant decrease in tumour size after 8 weeks. However, the tumours which were treated by RCF didn't show shrinkage. The difference was substantial. The tumors treated by RCF were able to show necrosis that is common in tumors that have been exposed to RCF. The possibility is that the necrosis was caused by a lack of oxygen in larger tumors.
In conclusion, the findings suggest the fact that AM EMF is a powerful source of anticancer effects in vitro and in the vivo. A number of studies have proven the fact that AM RF EMF produces measurable reduction in tumours for HCC patients. There is a possibility that AM RF EMF causes these effects because of CACNA1H which is a protein involved in the process of tissue-specific Ca2+ influx. Furthermore, AM RF EMF may exert a sustained influence on the development of HCC tumors in vivo.